Hi, I'm Priya!
I am a cellular and molecular neuroscientist focused on understanding the mechanisms of neurodegeneration and neurotoxicity. I am particularly interested in investigating how glial cells in the central nervous system, such as astrocytes, microglia, and oligodendrocytes, become dysfunctional and contribute to neurodegeneration.
Currently, I am a postdoctoral fellow in Shane Liddelow’s lab at NYU Grossman School of Medicine, where I am investigating reactive astrocytes in Alzheimer's disease (AD) and oligodendrocytes in X-linked Dystonia Parkinsonism. I'm grateful for The Leon Levy Foundation for partially supporting my research.
Before moving to New York, I pursued my PhD in Gaurav Chopra’s lab at Purdue University in the Chemistry Department. My thesis work, partially funded by the Eli-Lilly Stark Neurosciences Predoctoral Research Fellowship, focused on developing biochemical tools to evaluate microglial response to amyloid beta in the context of AD. I also characterized the proteome and lipidome of microglia and astrocytes, including lipid droplet-laden microglia and neurotoxic reactive astrocytes, and uncovered a mechanism behind lipid-mediated microglial dysfunction in AD.
My long-term goal is to establish a research program that advances our understanding of how glial cells contribute to neurodegeneration, with the aim of developing more effective therapeutic strategies for neurological disorders.
Currently, I am a postdoctoral fellow in Shane Liddelow’s lab at NYU Grossman School of Medicine, where I am investigating reactive astrocytes in Alzheimer's disease (AD) and oligodendrocytes in X-linked Dystonia Parkinsonism. I'm grateful for The Leon Levy Foundation for partially supporting my research.
Before moving to New York, I pursued my PhD in Gaurav Chopra’s lab at Purdue University in the Chemistry Department. My thesis work, partially funded by the Eli-Lilly Stark Neurosciences Predoctoral Research Fellowship, focused on developing biochemical tools to evaluate microglial response to amyloid beta in the context of AD. I also characterized the proteome and lipidome of microglia and astrocytes, including lipid droplet-laden microglia and neurotoxic reactive astrocytes, and uncovered a mechanism behind lipid-mediated microglial dysfunction in AD.
My long-term goal is to establish a research program that advances our understanding of how glial cells contribute to neurodegeneration, with the aim of developing more effective therapeutic strategies for neurological disorders.